Long-chain ceramides are cell non-autonomous signals linking lipotoxicity to endoplasmic reticulum stress in skeletal muscle | Nature Communications
Long-chain ceramides are cell non-autonomous signals linking lipotoxicity to endoplasmic reticulum stress in skeletal muscle | Nature Communications
A selective inhibitor of ceramide synthase 1 reveals a novel role in fat metabolism | Nature Communications
Long-chain ceramides are cell non-autonomous signals linking lipotoxicity to endoplasmic reticulum stress in skeletal muscle | Nature Communications
Frontiers | The Role of Ceramides in Insulin Resistance
Long-chain ceramides are cell non-autonomous signals linking lipotoxicity to endoplasmic reticulum stress in skeletal muscle | Nature Communications
Ceramides are early responders in metabolic syndrome development in rhesus monkeys | Scientific Reports
High-fat diet-induced upregulation of exosomal phosphatidylcholine contributes to insulin resistance | Nature Communications
Dihydroceramide- and ceramide-profiling provides insights into human cardiometabolic disease etiology | Nature Communications
Distinct mechanisms involving diacylglycerol, ceramides, and inflammation underlie insulin resistance in oxidative and glycolytic muscles from high fat-fed rats | Scientific Reports
AdipoR agonist increases insulin sensitivity and exercise endurance in AdipoR-humanized mice | Communications Biology
Plasma sphingolipids are biomarkers of metabolic syndrome in non-human primates maintained on a Western-style diet | International Journal of Obesity
Deletion of the diabetes candidate gene Slc16a13 in mice attenuates diet-induced ectopic lipid accumulation and insulin resistance | Communications Biology
Beta-cell specific Insr deletion promotes insulin hypersecretion and improves glucose tolerance prior to global insulin resistance | Nature Communications
IJMS | Free Full-Text | Sphingolipid Metabolism: New Insight into Ceramide-Induced Lipotoxicity in Muscle Cells
Long-chain ceramides are cell non-autonomous signals linking lipotoxicity to endoplasmic reticulum stress in skeletal muscle | Nature Communications
SMPDL3b modulates insulin receptor signaling in diabetic kidney disease | Nature Communications
Long-chain ceramides are cell non-autonomous signals linking lipotoxicity to endoplasmic reticulum stress in skeletal muscle | Nature Communications
The role of ceramides in metabolic disorders: when size and localization matters | Nature Reviews Endocrinology
Frontiers | Ceramides in Adipose Tissue
Frontiers | Adipocyte Ceramides—The Nexus of Inflammation and Metabolic Disease
IJMS | Free Full-Text | Ceramides as Mediators of Oxidative Stress and Inflammation in Cardiometabolic Disease
The aetiology and molecular landscape of insulin resistance | Nature Reviews Molecular Cell Biology
Long-chain ceramides are cell non-autonomous signals linking lipotoxicity to endoplasmic reticulum stress in skeletal muscle | Nature Communications
Ceramides are early responders in metabolic syndrome development in rhesus monkeys | Scientific Reports
Downregulation of lipin-1 induces insulin resistance by increasing intracellular ceramide accumulation in C2C12 myotubes
